My research aims at understanding the cellular mechanisms of complex cardiac arrhythmia diseases, such as heart failure and atrial fibrillation. This is achieved by developing mechanistic computational models, based on bioengineering principles. The purpose of my studies is to develop frameworks for accurate prediction of disease mechanisms, which is expected to lend insight into the origins of clinical observations and aid the identification of pro-arrhythmic targets. These models also serve as platforms for testing of pharmacological interventions to guide therapeutic strategies.

Accepting students per funding availability.